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The Healthy Herd

Before You Buy: Pre-purchase Considerations

February 19, 2021 by Kinder Goat Breeders Association

Purchasing goats is the exciting culmination of time and communication. Most buyers are familiar with typical pre-purchase considerations such as genetics, conformation, disease status and general health, and of course, location and price. Here are few additional things to consider before purchasing.

Climate

Changing locales and eventual introduction into a new herd is stressful and a drastic change in climate can increase stress. Goat breeds originated, with a some exceptions, in arid deserts or mountains. While goats can thrive in various climates, adjustment can take time. Genetic adaptation takes generations.

Excessive moisture, especially humidity, is not the ideal goat environment. Humid heat especially can cause heat stress in an animal unused to those conditions. Goats moving from dry, cool areas to humid, warm areas might initially benefit from additional cooling methods such as implementation of barn fans or buckets of cold water to lie against.

A goat sometimes has sparse or non-existent cashmere undercoat, making it particularly susceptible to the cold – this goat might be fine in the southern states but need extra attention if moved to an area with serious winters. Goats with inadequate cashmere coats might need extra calories, a safe heat source, or a blanket or coat.

The difference between a buyer’s climate and the seller’s climate probably won’t be such a serious consideration as to dissuade the buyer, but might tell the buyer to be extra-vigilant about the goat’s health until it grows accustomed to its new home.

General Management Practices

Goats are successfully raised across a wide spectrum of management styles; management varies greatly from producer to producer, from micro-managed operations to “survival of the fittest.” Most farms are somewhere in between the two extremes. It is wise to evaluate the general management practices of a producer compared to your own before purchasing, especially considering diet.

Generally speaking, it is easier for a goat to transition from a less-intensively managed operation than a very management-intense operation. For example, goats accustomed to free-choice alfalfa and large grain rations might lose condition when brought into a farm that primarily utilizes forage.

Conversely, goats unaccustomed to rich legumes or grain rations will need to be introduced to their new diet gradually. A sudden increase in these supplementations can lead to many health problems – slow changes are the key.

A goat used to walking a few steps to a buffet might lose condition when introduced to a farm where it is expected to traverse large areas or rough terrain. Goats in good body condition used to ranging might become over-conditioned when moved to a farm that utilizes dry-lotting and 24-7 availability of high-quality hay like alfalfa.

Most changes in condition because of a change in diet won’t be long-lasting, but the buyer should understand the goat’s current management to better care for the animal at its new farm.

Parasite Management

The most important consideration often over-looked is parasite management. When you buy the goat, you buy the worms, so the adage goes. If the new animal has internal parasites, especially Barber Pole worms, and is not dewormed prior to introduction into your herd, that animal will be shedding eggs in your pasture. Those eggs will hatch into larvae for your herd to pick up. Those larvae will mature and mate with worms already in your animals, passing on their genetic material.

It is possible to prevent initial egg shedding by deworming the animal with an effective product or products that show a 100% reduction rate in the fecal egg counts.  However, if you do not test the effectiveness of the treatment, or have no effective treatment, there is a very good chance a few worms will survive.

The impact of these surviving worms may or may not be serious, depending on a few things. If there is not a good kill rate, of course, the number of surviving worms will increase, as will their impact on your herd.


Ask the breeder what dewormer he or she uses.

By understanding anthelmentics classes, you can hypothesize if the goat is harboring parasites that will either be resistant or susceptible to the class of dewormer effective on your farm.

For example, in terms of strongyle treatment, the imidazothiazole Levamisol (one brand name is Prohibit®), is considered the most-effective class and the benzimidozoles or “white dewormers” the least effective. If the farm you are purchasing from already uses levamisol while you typically and effectively use the albendazole (which is a benzimidozole) Valbazen®, it is almost certain that the parasites in your new animal will be resistant to Valbazen®.

If you typically and effectively use ivermectin 1% and the seller typically uses moxidectin, it is possible that these parasites will not be effectively killed with ivermectin, even though both moxidectin and ivermectin are in the macrocyclic lactone class. Moxidectin is more potent.

For a great overview of classes, read “Choosing the Right Drug for Worm Control” by Lisa Williamson DVM, MS, DACVIM. The full text is available at the American Consortium for Small Ruminant Parasite Control website.

There are a few studies indicating that, for a short time, it is possible that bringing in susceptible parasites (i.e. the seller successfully deworms with a white dewormer while you use ivermectin) will dilute resistant parasite populations. Of course, this depends on several factors, and has only been successfully implemented in more intensively-managed grazing operations. The effects of this dilution were generally short-lived – a study where resistance was measured over 3 years later showed anthelmentic failure again – but the subject is still being researched. For more information, check out “Replacement of Resistant Worms with Susceptible Worms – Can We Do It, and Is It Sustainable?” by Melissa George, BS, MS at WormBoss.com.

Ask for deworming records and methods.

  • How often has the herd been dewormed?
  • How often has the individual goat you are considering purchasing required treatment?

Some farms accept a higher number of treatments year; others are quite strict and cull animals that need repeated treatments, especially in comparison to herdmates. A few do not tolerate an animal that needs dewormed, period.

To slow anthelmentic resistance, a general rule of thumb is animals should not need dewormed more than three times a year.

If the breeder chemically deworms on a schedule, seriously consider passing on that animal. This outdated practice is proven to very strongly select for resistant parasites and is one of the causes of complete anthelmentics failure in some farms – this means the parasites are resistant to every class of dewormer – and useless classes in others.

Remember that the best method for deworming is not to deworm on a schedule but by need. Does the seller deworm the entire herd or select only the animals that need treatment based on fecals, FAMACHA, and condition and thereby slow resistance and increase refugia?

For more information on dewormers and deworming practices, please refer to the blog posts “The Barber Pole Worm” and “Are You Deworming Your Goats Correctly?”

Ask about resistance and resilience of the sire and dam.

Studies have proven both resistance and resilience to parasite infections is moderately heritable. Breeding animals that require more-than-average treatments can negatively impact the herd’s genetics. Culling those less-hardy animals and breeding stock that requires fewer dewormings than the herd average can positively influence subsequent generations. It takes time, but it is a worthwhile effort to breed for animals that are more naturally tolerant or resistant to parasite burdens.

Will the new animal’s genes, in relation to parasite resistance or resilience, positively or negatively affect your herd?

Consider these few additional things before purchasing to better acclimate your new goat, improve or maintain the parasite resistant or resilient genes in your herd, or avoid purchasing an influx of tough-to-treat parasites.

By Kendra Shatswell

Filed Under: The Healthy Herd Tagged With: Management

Scruffy Coat Makes a Scruffy Goat

January 22, 2021 by Kinder Goat Breeders Association

When a Goat is Zinc Deficient

Kidding time is a wonderful time because it means new life on the farm! Who doesn’t enjoy watching these amazing creatures that can begin hopping and jigging within minutes of being born? It’s quite intriguing!

But what happens when, after days or weeks, mama begins showing signs of bodily stress? After all, she is giving all she’s got to producing amazing milk for those adorable kids. Sometimes trying to find what will help her can be daunting.

Let’s up her feed…that doesn’t work.
Let’s worm her…that doesn’t work.
Let’s try copper bolus…doesn’t work.
Well, how about selenium…nope.

What do you do when the most commonly mentioned stuff isn’t helping?

Well let’s consider our options. Upping the feed can get expensive. Worming over and over again creates resilient worms. Giving a stressed goat copper or selenium when those aren’t what is needed can be quite dangerous, because too much can lead to a potential overdose.

Let me introduce another player in the game of goat health: zinc. Such a tiny word and tiny mineral, but oh so important! Zinc deficiency, in my experience, shows up as very coarse hair, erratic shedding (even bald patches), and weight loss — symptoms are sometimes quite severe. Hoegger Farms uses the term “scruffy” to describe the appearance of a goat with zinc deficiency, and the word is quite fitting.

What causes zinc deficiency? There are a few possibilities.

First, an imbalance in the calcium to zinc intake ratio can result in a deficiency. If a goat is eating a diet or supplement high in calcium, that goat can become too low in zinc. Too much calcium blocks the efficient absorption of zinc. Or, alternatively, the goat’s diet may simply have too little zinc. Most US soils are zinc deficient to one degree or another, so zinc is an ingredient in a good goat mineral. Just decreasing the calcium intake may solve the issue, but sometimes going a step further really gets the ball rolling. I will tell you what we have chosen to do that has worked many times; but please note, I am not a professional. I am simply a goat mama who has learned what works for us through trial and error, talking to other goat owners, and my own research.

We decreased the alfalfa, since it is high in calcium. We then begin giving a human adult dose of zinc once a day. Typically, we can begin feeding our hungry mama more alfalfa after several days on zinc. Typically, within a month, we see a beautiful shiny coat and weight gain, but the transformation has happened as fast as two weeks. Goats in milk do require more food, so making sure does are getting enough is important. If that fails, give your scruffy doe’s calcium-to-zinc ratios some consideration. It’s cheap, safe, and simple.

Zinc deficiency can also crop up among other goats in your herd, including bucks. But the solutions are straightforward: check those feed ratios and supplement as required.

Healthy goats are happy goats, and happy goats make happy farms!

By Tamara Newton

Zinc deficiency (as well as copper toxicity) might also be caused by an imbalance in zinc to copper. A proposed ideal ratio is 4:1 zinc to copper.

Zinc deficiency symptoms include:
Dermatitis that is responsive to zinc supplementation.
Hair loss, especially on the back, legs, and face.
Weeping eyes.
Small testes and reduced libido in bucks
Hoof deformities, especially flaking hooves

After working closely with the Missouri Department of Agriculture, extensive testing on forage, hay, water, and mineral, and necropsies, it was determined an imbalance of zinc to copper was causing both secondary zinc deficiencies and copper toxicity in her Nigerian Dwarf herd. The buck pictured is exhibiting dermatitis and hair loss from secondary zinc deficiency. More information can be found at the website and Facebook page for Red Horse Valley, LLC and in the paper “In Search of Balance: A Nigerian Dwarf Breeder’s Experience with Chronic Copper Accumulation” written by the breeder, Kathy Winters.

Recently, research has also indicated that goats can exhibit zinc deficiency caused by genetics alone and might need additional oral zinc supplementation. Zinc can be supplemented in the form of loose minerals, lozenges, or capsules.

By Kendra Shatswell

Sources:
M.C. Smith and D.M. Sherman Goat Medicine, 2nd Ed

Zinc-responsive dermatosis in goats suggestive of hereditary malabsorption: two field cases. Krametter-Froetscher R, et al. Vet Dermatol. 2005.

Winters, K., 2019. In Search Of Balance: A Nigerian Dwarf Breeder’s Experience With Chronic Copper Accumulation.

Filed Under: The Healthy Herd

Goat Polio

January 8, 2021 by Kinder Goat Breeders Association

Polioencephalomalacia
More commonly called “goat polio” or simply “thiamine deficiency,” polioencephalomalacia is a neurological disease characterized by brain swelling and tissue death – the word literally means “softening of the brain.” Ruminants harbor beneficial bacteria and protozoa that produce Vitamin B12 (cobalamin) and Vitamin B1 (thiamine) under normal environmental conditions. If this production is interrupted, thiamine deficiency can occur. To put it simply, the rumen must be kept happy to produce thiamine; thiamine is necessary for proper nerve conduction, and neurological signs present when thiamine is lacking.
Pre-ruminant goat kids rely on dietary thiamine – goat milk and colostrum contains B vitamins. Low-quality colostrums or milk replacers or whole milk recipes lack the essential B vitamins for optimal kid health. Many factors can contribute to reduced or cessation of normal thiamine production in ruminants.

Dietary Causes

Dietary causes include too much grain and not enough roughage, consumption of moldy feedstuffs, diets high in sulfur, cobalt-deficient diets, or consuming certain toxic plants – such as bracken ferns.

Sudden changes in the diet, especially increased or excessive grain consumption, can decrease the PH in the rumen and disturb the thiamine production of the microorganisms there.  Excessive grain rations or moldy feedstuffs can be problematic because each contains certain enzymes that produce thiaminases. Thiaminases inactivate or degrade thiamine molecules, leaving the goat thiamine-deficient. Bracken ferns – while toxicity symptoms vary widely based on dose, duration, and species of affected animals – contain thiaminases, and thiamine deficiency has been noted in sheep grazing on the plants in Australia.

Diets high in sulfur – foodstuffs or water or a combination of the two – can also lead to thiamine deficiency. According to “Digestive System and Nutrient Needs of Meat Goats” by Purdue Extension, “Sulfur produces thiamine-like compounds called analogs that decrease the absorption of thiamine in the rumen.” There are many possible sources of excessive sulfur, including: water sources, alfalfa, cruciferous plants, distillers grains, and corn, sugar beet, and sugar cane byproducts. According to Goat Medicine by Mary C. Smith and David M. Sherman, “Extrapolating from recommendations based on cattle and sheep, the maximum total dietary sulfur in high concentrate diets is 0.30% and 0.05% if the diet consists of at least 40% forage. Drinking water should contain less that 600mg/L sulfate/L for high concentrate diets, whereas 2,500 mg sulfate/L is acceptable with higher forage intake (NRC 2005.)” Generally speaking, sulfur should not exceed 0.3% total diet dry matter.


Cobalt-deficient diets are also precursors to goat polio. The trace mineral is essential for the microorganisms to manufacture and utilize Vitamin B12. Vitamin B12 is essential to maintain nervous system integrity and promote red blood cell synthesis. Certain areas in the U.S. are known to cobalt-deficient – a map can be found at http://www2.luresext.edu/goats/training/nutrition.html. Sandy soils and alkaline soils are more likely to be cobalt-deficient. Though not much research has been conducted on goats specifically, it has been established that goats require a very small amount of cobalt and are less sensitive to cobalt deficiencies than sheep. Sufficient daily cobalt intake is between 0.1ppm and 0.3ppm. Toxicity can occur at 10ppm.

Additional Causes

Infectious and parasitic diseases increase thiamine requirements and can lead to goat polio if those requirements are not met. Since antibiotics do not distinguish between beneficial or harmful organisms, antibiotics can diminish the beneficial gut flora responsible for thiamine production.

Amprolium – the active ingredient in some coccidiostats such as CORID® – works by blocking thiamine uptake of certain coccidian protozoa that require more than the host. Misuse or long-term use can result in thiamine deficiency in the goat, especially if the goat experienced dietary thiamine deficiencies already.

Fortified Vitamin B Complex

Symptoms of Goat Polio

  • Off feed/water
  • Disorientation
  • Staggering
  • Circling
  • Stargazing
  • Convulsions
  • Involuntary eye movements
  • Temporary blindness
  • Sudden death

Note: Stargazing is characterized by the goat’s head thrown backwards due to rigid neck muscles. Temporary blindness may last as long as 2-3 weeks. Diarrhea, while not a neurological symptom, can present because the rumen is not functioning properly.

Treatment

Severe thiamine deficiency can possibly kill a goat in 24 hours to a few days if left untreated. Goats are more likely to die from poor rumen function or inability to eat. Correct treatment usually results in quick improvement. The only effective therapy is supplementing thiamine.

Very severe cases might require an intravenous treatment performed by a veterinarian. Fortified Vitamin B complex injections are more commonly used, and can be given subcutaneously or intramuscularly. The dosage will depend on the concentration of thiamine – for supplements containing 100mg of thiamine the common dosage is 1cc per 20lb. B Vitamins are water-soluble and excess is excreted through the urine.

Injections should be given every six hours as needed. Taper off treatments gradually, lengthening time between injections, to monitor for potential relapse. If the goat is truly experiencing goat polio, improvements can be seen in as little as minutes or hours. If no improvement is noticeable, consider consulting your vet about treating simultaneously for Listeriosis, which has very similar symptoms but is more common in adult animals. Read more about Listeriosis at Merck Veterinary Manual online.

By Kendra Shatswell


Sources

Article reviewed by Dr. Ken Brown DVM

“Animal Sciences Common Diseases and Health Problems in Sheep and Goats” – Purdue Extensions.

“Digestive System and Nutrient Needs of Meat Goats” – Purdue Extensions.

Articles.extension.org/pages/73364/goat-nutrition-cobalt.

Articles.extension.org/pages/19362/goat-nutrition-micronutrients.

https://www.dsm.com/markets/anh/en_US/Compendium/ruminants/thiamin.html

“Dietary Sulphur in Ruminant Diets” – Westway Feed Products

“Polioencephalomalacia (Goat Polio)” Alabama and Auburn Universities UNP-65 – Maria Leite-Browning, DVM, MS Extension Anima Scientist Alabama A&M University.

“Ramifications of Thiamine Deficiency” – Department of Natural Resources, Cornell University, 2017.

https://www.merckvetmanual.com/

Goat Medicine Mary C. Smith and David M. Sherman.

Filed Under: The Healthy Herd

Parasite Management – Are You Deworming Your Goats Correctly?

November 27, 2020 by Kinder Goat Breeders Association

Parasite management is crucial to a healthy, happy goat. The goal is a low-enough parasite burden animal health is not compromised. While there is not a singular parasite management plan that works for every farm, there are a few practices every owner should follow that will ensure goats are dewormed correctly and prolong the efficacy of chemical dewormers. Additionally, every goat owner should be aware of parasite resistance, and strive to postpone it as long as possible. “Anthelmintic resistance is defined as a heritable genetic change in a population of worms that enables some individual worms to survive drug treatments that are generally effective against the same species and stage of infection at the same dose rate,” according to “Biology of Anthelmintic Resistance: These Ain’t Your Father’s Parasites” by Dr. Ray M. Kaplan.


DEWORMERS

First, owners need to understand there are different classes of dewormers, also referred to as anthelmintics. A class consists of dewormers that share a similar mode of action. Many dewormers are off-label – not approved for goats without a valid relationship with a veterinarian. For a handy chart on dewormers, complete with dosages and crucial notes, visit https://www.wormx.info/dewormers or search the files at Goat Vet Corner group on Facebook.

The first class is benzimidazoles, also called the “white dewormers.” Dewormers in this class include fenbendazoles, albendazoles, and oxybendazole. Since this class has been around the longest, stomach worms are often resistant to these dewormers. Remember that resistance varies from farm to farm.  Fenbendazoles are effective against tapeworms and albendazoles are effective against adult liver flukes.

The second class is the macrocyclic lactones, which include avermectins and milbemycines. According to “Choosing the Right Drug for Worm Control” by Dr. Lisa Williamson – “Avermectins include ivermectin, eprinomectin, and doramectin. Moxidectin, a milbemycin, is chemically very similar to the avermectins…Moxidectin is a more potent, lipophilic macrocyclic lactone than ivermectin.” Though they are in the same class, moxidectin can still be effective where ivermectin is not.

The third class is the Imidazothiazole/tetrahydropyrimidine class. Common class members include levamisol and mortanel. Levamisol, while it is the easiest to overdose and MUST be carefully dosed by weight, is often the most effective class currently available in the United States.

DRUG CLASSDRUGCOMMON CLASS MEMBERS
Benzimidazolesfenbendazoles, albendazoles, oxybendazolesSafeguard, Panacur
Valbazen
Macrocyclic lactonesivermectins, moxidectin, doramectin, eprinomectinIvomec, Quest, Cydectin, Dectomax, Eprinex
Imidazothiazole/tetrahydropyrimidinelevamisol, mortanel tartrate,Prohibit, Rumatel

RULE ONE

Do NOT rotate dewormers.

Rotating between classes and dewormers leads to parasite resistance and accelerates dewormer failure. Find what dewomer works on your farm based on fecal egg count reduction tests – determine the egg count prior to deworming compared to fecal egg count 10-14 days after deworming. An effective dewormer will have at least a 95% kill rate. Dewormers can still be useful with a lower kill rate, but it might be a good idea to either combine two classes or use in conjunction with another alternative treatment, such as copper oxide wire particles or a diet high in tannins.

 A DrenchRite® test is also available that determines what parasites your herd is burdened with and the effectiveness of each dewormer class on the parasites. At the time this article is written, the DrenchRite® test cost $450.
Dewormers from two or more classes can be combined to deworm new animals, or prolong the life of dewormers when resistance has been noted or a dewormer class has failed. Note that these are given at the same time, but NOT mixed together. It should be cautioned that improper use of combination dewormers could select for worms that are resistant to ALL anthelmintics. For more information on combining dewormers and the DrenchRite® test, please refer to the following: https://www.wormx.info/combinations and https://www.wormx.info/drenchriteassay.

RULE TWO

Do NOT deworm on a schedule.

Contrary to recommendations given when dewormers first became available, deworming should not be scheduled. We now know scheduling treatment accelerates resistance, and chemical deworming should NOT be preventive. Parasite numbers wax and wane during different seasons, and in different conditions. Lactating does will likely be more susceptible to parasite burdens than a dry doe, and bucks are more likely to be susceptible in rut, etc….As detailed in the “Barber Pole Worm” article in the Winter 2017 newsletter, Barber Pole worms and other internal parasites thrive in certain conditions and will have lower survival rates in others. For example, Barber Pole worm eggs and larvae survive very well in warm, humid conditions, but use up their energy stores very quickly in dry heat. Learn about the life cycle and preferred conditions of the parasites your herd is dealing with to better manage them – Goat Biology.com has some excellent animated slides detailing the lives of common goat parasites. The American Consortium for Small Ruminant Parasite Control offers many articles and files on the subject, as well.

RULE THREE

Deworm only the animals that require treatment.

By selecting only animals that need deworming versus deworming the entire herd, the goat-keeper does not expose all worms to the chemical, thereby slowing the worm’s ability to develop genes to resist the anthelmentic. The idea of leaving unexposed parasites is called refugia. The Latin term means “in refuge.” By leaving untreated animals and therefore unexposed worms, there will be few resistant worms to mate with many susceptible worms, and the resistant genes will be diluted in the next generation. Refugia refers both to adult worms not exposed to dewormer and the larvae and eggs on pasture that were not the product of resistant worms.

Furthermore, it is documented that the parasite burden is NOT evenly distributed among the herd. Research shows 20-30% of goats in a herd will carry up to 80% of all parasites. By first identifying, then deworming and/or culling these animals, the goat owner will greatly reduce the parasite burden both in the goats and on the pasture.

Deworm animals based on FAMACHA scores – remember this only indicates anemia that is generally caused by Barber Pole worms or liver flukes – the Five Point system, and/or a fecal showing a high eggs per gram count. Many producers misuse the FAMACHA system – check out https://www.wormx.info/dosdonts.

 Fecals are crucial to determine what parasites your goats are burdened with, how heavy that burden is, and how well your dewormer and deworming practices are working. Utilize your livestock veterinarian, send in samples to the Veterinary Parasitology Laboratory (http://www.midamericaagresearch.net) or invest in your own microscope. Tolerable parasite levels will vary from farm to farm and goat to goat. Remember that some goats are more resilient to parasite burdens than others, but these animals are shedding higher numbers of eggs in the pasture.

Fecal Slide

RULE FOUR

Dose correctly.

Dose by weight to avoid under-dosing. WEIGH THE GOAT BEFORE DOSING. If you do not have access to a livestock scale, weight tapes or cloth measuring tapes are a cheap investment. Administer the dewormer correctly – from the article “Choosing the Right Drug for Worm Control” – “Delivery deep into the oral cavity avoids closure of the esophageal groove, so the medication goes into the rumen rather than the abomasum. This step facilitates longer contact time of the drug with the gastrointestinal tract and improves drug efficacy.”
Dewormers in the forms of pour-ons, injections, and long-range (persistent activity) dewormers have proved to be generally ineffective in goats. These sub-lethal levels mean many worms survive and develop those dreaded resistant genes. For further reading, check out “Should I consider using LongRange™ dewormer for parasite control in small ruminants?” by Dr. Ray Kaplan.

Fasting the animal 12-24 hours before treating with ivermectins and/or benzimadazoles is a good idea, with the exception of does in late gestation. This increases drug availability, meaning the worms will be exposed to the drug for longer and are thus more likely to be killed.

ADDITIONAL CONSIDERATIONS

As goat producers, we are exercising parasite management, not parasite elimination.

Dr. David Fernandez explained in an online parasite information session, “No matter how hard you try, you cannot eliminate every parasite from your animals. If you try hard enough, you will create some of the toughest parasites on Earth which will eventually make your life miserable, not to mention your goats’. Instead, we need to focus on managing parasites so that they do not cause an intolerable level of harm.”  In conclusion, managing – not eliminating – parasites will vary from farm to farm but ALWAYS dose correctly and treat only animals the need treated, and NEVER rotate dewormers or deworm on a schedule. Utilize tools like fecals and FAMACHA to determine which animals need treated and to determine effectiveness of the treatment.

SOURCES

  • “Biology of Anthelmintic Resistance: These Ain’t Your Father’s Parasites”; Ray M. Kaplan, DVM, PhD, DipACVM, DipEVPC College of Veterinary Medicine University of Georgia, Athens, GA
  • “Choosing the Right Drug for Worm Control”; Lisa Williamson DVM, MS, DACVIM University of Georgia College of Veterinary Medicine Athens, Georgia, USA
  • “Correct Administration of Anthelmentics” Ken Pettey Department of Production Animal Studies, Gareth Bath Department of Production Animal Studies, Jan van Wyk Department of Veterinary Tropical Diseases, Faculty of Veterinary Science, University of Pretoria.
  •  “Do’s and Don’ts of FAMACHA Scoring©” Katherine Petersson, PhD Associate Professor of Animal Science, University of Rhode Island
  • “Extending the Efficacy of Anthelmintics”; Lisa H Williamson, DVM, MS, DACVIM University of Georgia College of Veterinary Medicine
  •  “Goat and Sheep Parasite Control” (powerpoint presentation) Jerry Lamb, University of Tennessee Extension, Rhea County.
  • “Refugia – Overlooked as Perhaps the Most Potent Factor Concerning the Development of Anthelmintic Resistance” [2001] Van Wyk, J.A. (Pretoria Univ., Onderstepoort (South Africa). Veterinary Tropical Diseases Dept.)
  •  “Should I consider using LongRange™ dewormer for parasite control in small ruminants?”; by Dr. Ray Kaplan Professor of Parasitology, Department of Infectious Diseases College of Veterinary Medicine University of Georgia Athens, Georgia

By Kendra Shatswell

Filed Under: The Healthy Herd Tagged With: Deworming, Parasite Management

Preparing Your Does For Breeding

September 27, 2012 by Kinder Goat Breeders Association

For many goat owners, fall is breeding season – a busy and exciting time around the farm!
If you are planning on breeding your does any time soon, they should be in top form. This means making sure they are in good weight (see previous article on body scoring your goats), free of heavy parasite loads and ready to start making strong, healthy babies. Below are some other things that should be considered before breeding…

Healthy goats start with healthy feet. Your goats should be on a regular trimming schedule, and making sure feet are regularly trimmed during pregnancy is more important than at any other time. Because Kinders often have multiple kids, they carry a large amount of weight throughout pregnancy. Leaving hooves untrimmed during this time can cause irreversible problems. Hooves often grow faster during pregnancy, so closely inspect your goats’ feet every week.

Goats should always have access to free choice minerals, but some areas are so deficient in certain minerals that they can not be completely replaced by free choice minerals. In those areas, extra supplements need to be given during pregnancy to avoid health problems with does and newborn kids. If you are not sure whether or not your area is deficient in anything, ask your local veterinarian.

Finally, stand back and look at your goats. Consider how they look and act – do you have nagging concerns about any of them? Does something seem a little bit off in a certain doe? Now is the time to treat any issues you think might be present, rather than risking the possibility that they worsen or become untreatable while your doe is pregnant. Cutting corners on herd health can cause major problems with your does, their births and their kids.

Preparing your goats for breeding season is easy, and the benefits are huge. By covering these few basic items, you will ensure a happier, healthier breeding season for your entire herd, and more time to enjoy those sweet little kids when they finally show up!

Filed Under: The Healthy Herd

Dehorning

September 9, 2012 by Kinder Goat Breeders Association

http://www.goatwisdom.com/Video/Dehorning.html

 

We apply a paste call nu-stock after we dehorn.

Filed Under: The Healthy Herd

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